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Atorvastatin patient uk raine, n=538) or an older, healthy person (not age-related) was randomly assigned to receive either 600mg of lovastatin dissolved in 150 mL of water (n=541), or placebo (n=543) for 8 weeks. Statistical Analysis Baseline characteristics of groups were measured by the Fisher exact test. We performed stratified analyses of propensity scores to determine which patients, if any, would be more likely to randomly assigned receive lovastatin or placebo and to assess the differences between groups. After adjusting buy atorvastatin 10 mg the propensity scores for Atorva 100 Capsules 200mg $199 - $1.99 Per pill baseline severity, age, sex, hypertension, history of myocardial infarction, and cholesterol level, mean difference in total cholesterol Atorvastatin calcium generic sandoz was 0.1 mm, with the 95% confidence limits as follows: 0.1-0.2 mm (P=.06); 0.2-0.3 (P=.002); and 0.3-0.5 mm (P=.001); mean change in HDL cholesterol the patients lovastatin group was 1.5%. Mean changes from baseline to 12 weeks were −13.6 and −8.4 mg/dL in the patients lovastatin and placebo groups, respectively (P<.001 vs. baseline). The mean change from baseline to 12 weeks in VLDL+ LDL cholesterol ratio was −0.6; −0.7 in the patients lovastatin and placebo groups, respectively. Because the primary outcome measure was clinical progression of disease and therefore the effect size is expected to be small in subgroups with different baseline characteristics, we assessed the ability of propensity scores to generate a clinically meaningful effect size between groups. For the primary efficacy endpoint (clinical progression of disease), the differences in change from baseline at 12 weeks between the mean propensity score difference at baseline between groups were as follows: 0.02 for the lovastatin versus placebo group; 0.04 for the lovastatin versus healthy comparison (non-smoking adult without a history of hypertension); and 0.01 for the lovastatin versus healthy comparison (not a patient with history of myocardial infarction or any other cardiovascular risk factors in primary care or whom a objective of care was to improve the lipid profile). propensity scores demonstrated low sensitivity to detect clinically meaningful effects, even when comparing two groups with identical baseline characteristics. The relationship between baseline parameters of interest (diet, weight, total cholesterol, and HDL cholesterol) clinical effects is shown in Figure 1. After adjustment for these covariates, the mean difference in change from baseline was −0.9 mm in the patients lovastatin group, with a 95% confidence limit as follows: −1.1 mm (P=.001); −2.5 and −0.6 (P=.09). After adjustment for baseline atorvastatin 20 mg coupons severity and lipid variables stratification by patients with a primary objective of care to improve lipid profile, the mean difference in change from baseline was −4.5 mm in the patients lovastatin group, with a 95% confidence limit as follows: −8.7 mm (P<.001); −2.6 (P=.006); and −0.6 mm (P=.16). Effect size was calculated based on the percentage of patients randomized to receive lovastatin who experienced clinical progression of the primary end point. effect size for this primary end point was 0.2 mm (95% confidence limit, −0.2 mm, or 1/1,000 patients). The effect size for secondary end point of clinical progression disease was calculated as −0.3mm (or −0.6mm with stratification for patients a primary objective of care to improve lipid profile);

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